The Roles of protein glycation, glycoxidation, and advanced glycation end-product formation in diabetes-induced atherosclerosis

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dc.contributor.author Rashid, I
dc.contributor.author Brown, BE
dc.contributor.author Van Reyk, DM
dc.contributor.author Davies, MJ
dc.date.accessioned 2010-06-16T04:55:47Z
dc.date.issued 2006
dc.identifier.citation Biochemistry of Atherosclerosis, 2006, pp. 247 - 283
dc.identifier.isbn 0387312528
dc.identifier.isbn 9780387312521
dc.identifier.other B1 en_US
dc.identifier.uri http://hdl.handle.net/10453/11659
dc.description.abstract Diabetes is known to induce a range of micro-and macrovascular complications, with the latter resulting in premature and accelerated atherosclerosis. Thus people with diabetes have a 2-4-fold increased risk of developing cardiovascular diseases which is responsible for ca. 50% of deaths amongst people with diabetes. The mechanisms behind this elevated risk are still not fully understood, though there is now increasing evidence for a role of glycation and glycoxidation reactions induced by hyperglycemia. This article reviews current knowledge of the role that these reactions play in diabetesinduced atherosclerosis with particular emphasis on the molecular reactions that result in the modification of lipoproteins, and the consequences of these reactions on cellular metabolism. © 2006 Springer Science+Business Media, LLC. All rights reserved.
dc.publisher Springer US
dc.relation.isbasedon 10.1007/0-387-36279-3_12
dc.title The Roles of protein glycation, glycoxidation, and advanced glycation end-product formation in diabetes-induced atherosclerosis
dc.type Chapter
dc.parent Biochemistry of Atherosclerosis
dc.journal.number en_US
dc.publocation New York, USA en_US
dc.identifier.startpage 247 en_US
dc.identifier.endpage 283 en_US
dc.cauo.name FEIT. A/DRsch Ctre for Health Technologies en_US
dc.conference Verified OK en_US
dc.for 110201 Cardiology (Incl. Cardiovascular Diseases)
dc.personcode 000788
dc.percentage 100 en_US
dc.classification.name Cardiology (incl. Cardiovascular Diseases) en_US
dc.classification.type FOR-08 en_US
dc.edition 1 en_US
dc.custom en_US
dc.date.activity en_US
dc.location.activity en_US
dc.description.keywords advanced glycation end products
dc.description.keywords aldehydes
dc.description.keywords atherosclerosis
dc.description.keywords carbonyl stress
dc.description.keywords cholesterol
dc.description.keywords cholesterol esters
dc.description.keywords foam cell
dc.description.keywords glucose
dc.description.keywords glycation
dc.description.keywords glycoxidation
dc.description.keywords lipoprotein
dc.description.keywords macrophages
dc.description.keywords oxidation
pubs.embargo.period Not known
pubs.organisational-group /University of Technology Sydney
pubs.organisational-group /University of Technology Sydney/Faculty of Science
utslib.copyright.status Closed Access
utslib.copyright.date 2015-04-15 12:17:09.805752+10
pubs.consider-herdc true
utslib.collection.history School of Medical and Molecular Sciences (ID: 341)
utslib.collection.history Closed (ID: 3)
utslib.collection.history School of Medical and Molecular Sciences (ID: 341)
utslib.collection.history School of Medical and Molecular Sciences (ID: 341)


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