TNF and leptin tell essentially the same story in alzheimer's disease

Clark, IA; Alleva, LM; Vissel, B

TNF and leptin tell essentially the same story in alzheimer's disease

Clark, IA Alleva, LM Vissel, B

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Publication Type:: Journal Article
Citation:: Journal of Alzheimer's Disease, 2011, 26 (2), pp. 201 - 205
Issue Date:: 2011-01-01

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Field	Value	Language
dc.contributor.author	Clark, IA	en_US
dc.contributor.author	Alleva, LM	en_US
dc.contributor.author	Vissel, B https://orcid.org/0000-0001-8860-8050	en_US
dc.date.issued	2011-01-01	en_US
dc.identifier.citation	Journal of Alzheimer's Disease, 2011, 26 (2), pp. 201 - 205	en_US
dc.identifier.issn	1387-2877	en_US
dc.identifier.uri	http://hdl.handle.net/10453/117783
dc.description.abstract	Both tumor necrosis factor (TNF) and leptin are, independently, under investigation as the central mechanism for Alzheimer's disease. The wider literature provides every indication that both mediators are part of the same pathways thought to cause functional loss in this condition. This association, which has not been specifically addressed in the Alzheimer's disease literature, may be a useful link to expedite future study into the pathogenesis of this condition. © 2011-IOS Press and the authors. All rights reserved.	en_US
dc.relation.ispartof	Journal of Alzheimer's Disease	en_US
dc.relation.isbasedon	10.3233/JAD-2011-110266	en_US
dc.subject.classification	Neurology & Neurosurgery	en_US
dc.subject.mesh	Humans	en_US
dc.subject.mesh	Alzheimer Disease	en_US
dc.subject.mesh	Leptin	en_US
dc.subject.mesh	Tumor Necrosis Factor-alpha	en_US
dc.title	TNF and leptin tell essentially the same story in alzheimer's disease	en_US
dc.type	Journal Article
utslib.citation.volume	2	en_US
utslib.citation.volume	26	en_US
utslib.for	1103 Clinical Sciences	en_US
utslib.for	1109 Neurosciences	en_US
utslib.for	1702 Cognitive Sciences	en_US
pubs.embargo.period	Not known	en_US
pubs.organisational-group	/University of Technology Sydney
pubs.organisational-group	/University of Technology Sydney/Faculty of Science
pubs.organisational-group	/University of Technology Sydney/Faculty of Science/School of Life Sciences
utslib.copyright.status	closed_access
pubs.issue	2	en_US
pubs.publication-status	Published	en_US
pubs.volume	26	en_US

Abstract:

Both tumor necrosis factor (TNF) and leptin are, independently, under investigation as the central mechanism for Alzheimer's disease. The wider literature provides every indication that both mediators are part of the same pathways thought to cause functional loss in this condition. This association, which has not been specifically addressed in the Alzheimer's disease literature, may be a useful link to expedite future study into the pathogenesis of this condition. © 2011-IOS Press and the authors. All rights reserved.

Please use this identifier to cite or link to this item:

http://hdl.handle.net/10453/117783