Clonal diversity in carcinomas: its implications for tumour progression and the contribution made to it by epithelial-mesenchymal transitions

Lyons, J; Lobo, E; Martorana, A; Myerscough, M

Clonal diversity in carcinomas: its implications for tumour progression and the contribution made to it by epithelial-mesenchymal transitions

Lyons, J Lobo, E Martorana, A Myerscough, M

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Publisher:: Springer
Publication Type:: Journal Article
Citation:: Lyons J et al. 2008, 'Clonal diversity in carcinomas: its implications for tumour progression and the contribution made to it by epithelial-mesenchymal transitions', Springer, vol. 25, no. 6, pp. 665-677.
Issue Date:: 2008

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Field	Value	Language
dc.contributor.author	Lyons, J	en_US
dc.contributor.author	Lobo, E	en_US
dc.contributor.author	Martorana, A	en_US
dc.contributor.author	Myerscough, M	en_US
dc.date.accessioned	2012-02-02T11:01:23Z
dc.date.available	2012-02-02T11:01:23Z
dc.date.issued	2008	en_US
dc.identifier	2008000220	en_US
dc.identifier.citation	Lyons J et al. 2008, 'Clonal diversity in carcinomas: its implications for tumour progression and the contribution made to it by epithelial-mesenchymal transitions', Springer, vol. 25, no. 6, pp. 665-677.	en_US
dc.identifier.issn	0262-0898	en_US
dc.identifier.other	C1	en_US
dc.identifier.uri	http://hdl.handle.net/10453/15640
dc.description.abstract	The progression of tumours to malignancy is commonly considered to arise through lineal evolution, a process in which mutations conferring pro-oncogenic cellular phenotypes are acquired by a succession of ever-more dominant clones. However, this model is	en_US
dc.publisher	Springer	en_US
dc.relation.ispartof	Verified OK	en_US
dc.relation.ispartof	Clinical & Experimental Metastasis	en_US
dc.relation.isbasedon	http://dx.doi.org/10.1007/s10585-007-9134-2	en_US
dc.subject	Human Breast-Carcinoma; Mouse Mammary-Tumors; Collagen Gel Cultures; Basal-Cell Carcinoma; B-16 Melanoma-Cells; Tissue Reconstruction; Extracellular-Matrix; Cadherin Expression; Somatic Mosaicism; Dissociated Cells	en_US
dc.subject.classification	Clinical Sciences	en_US
dc.title	Clonal diversity in carcinomas: its implications for tumour progression and the contribution made to it by epithelial-mesenchymal transitions	en_US
dc.type	Journal article
utslib.citation.volume	25	en_US
utslib.citation.number	6	en_US
utslib.location	Dordrecht, The Netherlands	en_US
utslib.citation.startpage	665	en_US
utslib.citation.endpage	677	en_US
utslib.identifier.org	SCI.Faculty of Science	en_US
utslib.for	110300	en_US
utslib.percentage	100	en_US
dc.location.activity	ISI:000257719800009	en_US
utslib.copyright.status	closed_access
pubs.declined	1970-01-01T00:00:00.0+1000

Abstract:

The progression of tumours to malignancy is commonly considered to arise through lineal evolution, a process in which mutations conferring pro-oncogenic cellular phenotypes are acquired by a succession of ever-more dominant clones. However, this model is

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http://hdl.handle.net/10453/15640