Is there an association between the level of ambient air pollution and COVID-19?
- Publisher:
- AMER PHYSIOLOGICAL SOC
- Publication Type:
- Journal Article
- Citation:
- American journal of physiology. Lung cellular and molecular physiology, 2020, 319, (3), pp. L416-L421
- Issue Date:
- 2020-09
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ajplung.00244.2020.pdf | Published version | 892.13 kB |
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Full metadata record
Field | Value | Language |
---|---|---|
dc.contributor.author |
Wang, B https://orcid.org/0000-0002-8701-3896 |
|
dc.contributor.author |
Chen, H https://orcid.org/0000-0001-6883-3752 |
|
dc.contributor.author | Chan, YL | |
dc.contributor.author | Oliver, BG | |
dc.date.accessioned | 2020-12-17T01:11:23Z | |
dc.date.available | 2020-12-17T01:11:23Z | |
dc.date.issued | 2020-09 | |
dc.identifier.citation | American journal of physiology. Lung cellular and molecular physiology, 2020, 319, (3), pp. L416-L421 | |
dc.identifier.issn | 1522-1504 | |
dc.identifier.issn | 1522-1504 | |
dc.identifier.uri | http://hdl.handle.net/10453/144789 | |
dc.description.abstract | Epidemiological studies suggest that environmental factors (e.g., air pollution) can influence the spread and infectivity of coronavirus disease 2019 (COVID-19); however, very few papers have investigated or discussed the mechanism behind the phenomenon. Given the fact that pollution will increase as social distancing rules are relaxed, we summarized the current understanding of how air pollution may affect COVID-19 transmission and discussed several possible mechanisms. Air pollution exposure can dysregulate the human immune response and make people more susceptible to infections, and affect infectivity. For example, in response to exposure to air pollution, angiotensin-converting enzyme 2 will increase, which is the receptor for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). This may increase the efficiency of viral infection. It is also possible that air pollution can facilitate SARS-CoV-2 spread by increasing the transmission, and potentially, SARS-CoV-2 can also survive longer when attached to a pollutant. | |
dc.format | Print-Electronic | |
dc.language | eng | |
dc.publisher | AMER PHYSIOLOGICAL SOC | |
dc.relation | http://purl.org/au-research/grants/nhmrc/APP1158186 | |
dc.relation | Maridulu Budyari Gumal - The Sydney Partnership for Health, Education, Research and Enterprise (SPHERE)N/A | |
dc.relation | http://purl.org/au-research/grants/nhmrc/APP1079184 | |
dc.relation | http://purl.org/au-research/grants/nhmrc/GNT1158186 | |
dc.relation | http://purl.org/au-research/grants/nhmrc/GNT1079184 | |
dc.relation | http://purl.org/au-research/grants/nhmrc/1158186 | |
dc.relation.ispartof | American journal of physiology. Lung cellular and molecular physiology | |
dc.relation.isbasedon | 10.1152/ajplung.00244.2020 | |
dc.rights | info:eu-repo/semantics/closedAccess | |
dc.subject | 0606 Physiology, 1116 Medical Physiology | |
dc.subject.classification | Respiratory System | |
dc.subject.mesh | Humans | |
dc.subject.mesh | Coronavirus | |
dc.subject.mesh | Pneumonia, Viral | |
dc.subject.mesh | Coronavirus Infections | |
dc.subject.mesh | Peptidyl-Dipeptidase A | |
dc.subject.mesh | Air Pollution | |
dc.subject.mesh | Disease Transmission, Infectious | |
dc.subject.mesh | Pandemics | |
dc.subject.mesh | Betacoronavirus | |
dc.subject.mesh | Humans | |
dc.subject.mesh | Coronavirus | |
dc.subject.mesh | Pneumonia, Viral | |
dc.subject.mesh | Coronavirus Infections | |
dc.subject.mesh | Peptidyl-Dipeptidase A | |
dc.subject.mesh | Air Pollution | |
dc.subject.mesh | Disease Transmission, Infectious | |
dc.subject.mesh | Pandemics | |
dc.subject.mesh | Betacoronavirus | |
dc.subject.mesh | Air Pollution | |
dc.subject.mesh | Angiotensin-Converting Enzyme 2 | |
dc.subject.mesh | Betacoronavirus | |
dc.subject.mesh | COVID-19 | |
dc.subject.mesh | Coronavirus | |
dc.subject.mesh | Coronavirus Infections | |
dc.subject.mesh | Disease Transmission, Infectious | |
dc.subject.mesh | Humans | |
dc.subject.mesh | Pandemics | |
dc.subject.mesh | Peptidyl-Dipeptidase A | |
dc.subject.mesh | Pneumonia, Viral | |
dc.subject.mesh | SARS-CoV-2 | |
dc.title | Is there an association between the level of ambient air pollution and COVID-19? | |
dc.type | Journal Article | |
utslib.citation.volume | 319 | |
utslib.location.activity | United States | |
utslib.for | 0606 Physiology | |
utslib.for | 1116 Medical Physiology | |
pubs.organisational-group | /University of Technology Sydney/Faculty of Science | |
pubs.organisational-group | /University of Technology Sydney/Strength - CHT - Health Technologies | |
pubs.organisational-group | /University of Technology Sydney/Faculty of Science/School of Life Sciences | |
pubs.organisational-group | /University of Technology Sydney | |
utslib.copyright.status | closed_access | * |
pubs.consider-herdc | false | |
dc.date.updated | 2020-12-17T01:11:19Z | |
pubs.issue | 3 | |
pubs.publication-status | Published | |
pubs.volume | 319 | |
utslib.citation.issue | 3 |
Abstract:
Epidemiological studies suggest that environmental factors (e.g., air pollution) can influence the spread and infectivity of coronavirus disease 2019 (COVID-19); however, very few papers have investigated or discussed the mechanism behind the phenomenon. Given the fact that pollution will increase as social distancing rules are relaxed, we summarized the current understanding of how air pollution may affect COVID-19 transmission and discussed several possible mechanisms. Air pollution exposure can dysregulate the human immune response and make people more susceptible to infections, and affect infectivity. For example, in response to exposure to air pollution, angiotensin-converting enzyme 2 will increase, which is the receptor for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). This may increase the efficiency of viral infection. It is also possible that air pollution can facilitate SARS-CoV-2 spread by increasing the transmission, and potentially, SARS-CoV-2 can also survive longer when attached to a pollutant.
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