beta(2)-Agonist Induced cAMP Is Decreased in Asthmatic Airway Smooth Muscle Due to Increased PDE4D
- Publisher:
- 22
- Publication Type:
- Journal Article
- Citation:
- Plos One, 2011, 6 (5)
- Issue Date:
- 2011-01
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Full metadata record
Field | Value | Language |
---|---|---|
dc.contributor.author | Trian, T | en_US |
dc.contributor.author | Burgess, J | en_US |
dc.contributor.author | Niimi, K | en_US |
dc.contributor.author | Moir, L | en_US |
dc.contributor.author | Ge, Q | en_US |
dc.contributor.author | Berger, P | en_US |
dc.contributor.author | Liggett, S | en_US |
dc.contributor.author | Black, J | en_US |
dc.contributor.author | Oliver, BG | en_US |
dc.date.issued | 2011-01 | en_US |
dc.identifier.citation | Plos One, 2011, 6 (5) | en_US |
dc.identifier.uri | http://hdl.handle.net/10453/35585 | |
dc.description.abstract | Background and Objective: Asthma is associated with airway narrowing in response to bronchoconstricting stimuli and increased airway smooth muscle (ASM) mass. In addition, some studies have suggested impaired beta-agonist induced ASM relaxation in asthma | en_US |
dc.publisher | 22 | en_US |
dc.relation.ispartof | Plos One | en_US |
dc.relation.isbasedon | 10.1371/journal.pone.0020000 | en_US |
dc.subject.classification | General Science & Technology | en_US |
dc.subject.mesh | Muscle, Smooth | en_US |
dc.subject.mesh | Lung | en_US |
dc.subject.mesh | Myocytes, Smooth Muscle | en_US |
dc.subject.mesh | Humans | en_US |
dc.subject.mesh | Asthma | en_US |
dc.subject.mesh | Isoproterenol | en_US |
dc.subject.mesh | Cyclic AMP | en_US |
dc.subject.mesh | Phosphodiesterase Inhibitors | en_US |
dc.subject.mesh | Cell Proliferation | en_US |
dc.subject.mesh | Phenotype | en_US |
dc.subject.mesh | Cyclic Nucleotide Phosphodiesterases, Type 3 | en_US |
dc.subject.mesh | Adrenergic beta-2 Receptor Agonists | en_US |
dc.subject.mesh | Cyclic Nucleotide Phosphodiesterases, Type 4 | en_US |
dc.title | beta(2)-Agonist Induced cAMP Is Decreased in Asthmatic Airway Smooth Muscle Due to Increased PDE4D | en_US |
dc.type | Journal Article | |
utslib.citation.volume | 5 | en_US |
utslib.citation.volume | 6 | en_US |
utslib.for | MD Multidisciplinary | en_US |
pubs.embargo.period | Not known | en_US |
pubs.organisational-group | /University of Technology Sydney | |
pubs.organisational-group | /University of Technology Sydney/Faculty of Science | |
pubs.organisational-group | /University of Technology Sydney/Faculty of Science/School of Life Sciences | |
pubs.organisational-group | /University of Technology Sydney/Strength - CHT - Health Technologies | |
utslib.copyright.status | open_access | |
pubs.declined | 1970-01-01T00:00:00.0+1000 | |
pubs.consider-herdc | false | en_US |
pubs.issue | 5 | en_US |
pubs.volume | 6 | en_US |
Abstract:
Background and Objective: Asthma is associated with airway narrowing in response to bronchoconstricting stimuli and increased airway smooth muscle (ASM) mass. In addition, some studies have suggested impaired beta-agonist induced ASM relaxation in asthma
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