High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway

Blackwell Publishing
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Journal Article
Clinical And Experimental Pharmacology And Physiology, 2005, 32 (9), pp. 771 - 776
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1. Endothelial barrier dysfunction plays a pivotal role in the pathogenesis of diabetic vascular complications. The precise molecular mechanisms by which hyperglycaemia causes the increased permeability in endothelial cells are not yet well understood. I
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